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Ativan structure has been formulated in a way that it binds to the benzodiazepine receptors on the post-synaptic GABA-A channel. Basically, it helps with the increase in conductance of chloride ions by causing a shift into the cell for stabilization. This is achieved by enhancing the GABA receptors off its inhibitory effects. The inhibitory action of Ativan in two different places in the brain namely amyglada and cerebral cortex aids in smoothing the ends of anxiety and seizure disorders respectively.
